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02-27-2018, 02:13 AM
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At the age of 23 I have been on Hydrea for about two years now due to Essential Thrombocythemia and my platelets count over 2 million. I have tried many different medications for the same purpose but the majority of them caused unbearable side effects. And in spite of the fact that with Hydrea I do have some side effects - fatigue, some muscle pain and easy bruising - I can tolerate them and in addition I get so much better control over my platelets level what is a big change for me. My doctor helps me adjust my dosage and monitors my health condition. He also observes the positive effect Hydrea provides, so I am quite enthusiastic and hopeful. Would recommend Hydrea to other people with similar health condition.

[Image: 9gibbmj0s2egeg.gif]

I have essential thrombocytemia and have been on Hydrea for more than 5 years. The dosage differs from time to time, according to my blood tests. Actually I undergo blood tests every months since the time I was diagnosed with the disease. This is not the first thrombocytemia medication I am on, but this is by far the best one. With Hydrea I have least side effects and it provides excellent results on my platelets level. However I have some nausea and occasional headaches I feel much better with Hydrea than with any other med. I do hope that one day I will be able to find a more effective treatment that will help me conquer the illness but in general I am happy with Hydrea as it makes my quality of life so much higher. My doctor is also satisfied with the results we achieve when on Hydrea and he plans to continue this treatment course. I wish him lots of luck!)

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In SCD mice, some of the NO donated by HU is probably scavenged by cell-free hemoglobin, possibly explaining why its immediate NO-dependent effects require amplification by BAY73-6691 to achieve the full cGMP-elevating benefit in these mice.

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The mechanism accounting for this is unknown but might reflect the slower centripetal regression of red, or hematopoietic marrow to the axial skeleton in the presence of expanded erythropoiesis that is the result of sustained hemolysis.

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The frequency of episodes was relatively constant between 3 and 15 months and accounted for about half of the dactylitis episodes in the first 5 years of life in SS patients.

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We have recently reviewed our patients on hydroxyurea and found that there blood test parameters are much better.

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The sickle cells also block the flow of blood through vessels, resulting in lung tissue damage that causes acute chest syndrome, pain episodes, stroke and priapism (painful, prolonged erection).

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The role of this approach in the context of concomitant chemoradiotherapy is currently being investigated in several large, multicenter, randomized trials.

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One of those patients, 12-year-old Keone Penn, was apparently the first person to be cured of sickle-cell disease through this method.

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Now new research from the Johns Hopkins Children's Center and other institutions reveals that by preventing such complications, the drug can also considerably lower the overall cost of medical care in children with this condition.

In a previous open-label study of hydroxyurea therapy, the synthesis of fetal hemoglobin increased in most patients with sickle cell anemia, with only mild myelotoxicity.

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